The emails tell recipients that the passwords on their Facebook accounts have been reset, urging them to click on an attachment to obtain new login credentials, according to anti-virus software maker McAfee Inc. While individuals with little or no long-term virus had adaptive NK cell production, individuals with higher levels of virus had not only an absence of adaptive NK cells, but also a reduction in NK cell activity, the researchers added.New password-stealing Facebook virus could affect millions of usersīOSTON - Hackers have flooded the Internet with virus-tainted spam that targets Facebook’s estimated 400 million users in an effort to steal banking passwords and gather other sensitive information. The study sheds light on a mechanism that may explain the presence of viral reservoirs. The study shows that in some animals, macrophages infected with SARS-CoV-2 become resistant to destruction by NK cells, while in others, NK cells are able to adapt to infection (known as adaptive NK cells) and destroy resistant cells, in this case macrophages. "Yet it has long been known that NK cells play an important role in controlling viral infections," the researcher said. "The cellular response of innate immunity, which is the body's first line of defense, has been little studied in SARS-CoV-2 infections until now," said Muller-Trutwin. To understand the role of innate immunity in controlling these viral reservoirs, the scientists then turned their attention to NK (natural killer) cells. "What's more, we cultured these viruses and were able to observe, using the tools we developed to study HIV, that they were still capable of replicating," Huot said. "We were really surprised to find viruses in certain immune cells – alveolar macrophages – after such a long period and when regular PCR tests were negative," said Nicolas Huot, first author of the study and researcher in the Institut Pasteur's HIV, Inflammation and Persistence Unit. The researchers also found that the amount of persistent virus in the lungs was lower for the Omicron strain than for the original SARS-CoV-2 strain. Initial results from the study indicate that viruses were found in the lungs of some individuals six to 18 months after infection, even though the virus was undetectable in the upper respiratory tract or blood. The study analysed biological samples from animal models that had been infected by the virus. We therefore suspected that it could be due to the presence of the virus in the body," said Michaela Muller-Trutwin, Head of the Institut Pasteur's HIV, Inflammation and Persistence Unit. "We observed that inflammation persisted for long periods in primates that had been infected by SARS-CoV-2. It could also be the case for the SARS-CoV-2 virus which causes COVID-19, they said. This is the case for HIV, which remains latent in certain immune cells and can reactivate at any time. They remain in what are known as 'viral reservoirs', the researchers said. Some viruses persist in the body in a discreet and undetectable manner after causing an infection. The results, published in the journal Nature Immunology, show not only that SARS-CoV-2 is found in the lungs of certain individuals for up to 18 months after infection, but also that its persistence appears to be linked to a failure of innate immunity, the first line of defense against pathogens. Stay tuned with breaking news on HT Channel on Facebook.
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